Hypotheses
Health care practitioners who diagnose this syndrome explain that intestinal inflammation which may originate from intestinal dysbiosis or other sources of irritation, widens the junctions between the cells of the intestinal lining, allowing endotoxins and incompletely digested particles to be partially absorbed.
These are targeted by antibodies, forming immune complexes which cause a semi-infectious state and can be carried by the bloodstream to distant sites where they may stimulate the release of cytokines.
Low grade fever, transient gut pain, and a sense of inability to absorb nutrients are some reported symptoms in otherwise undiagnosed patients.
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